When viewed in the light of Gitt’s multidimensional theory of information, Darwinian evolution falls apart. The structure of life, thought by Darwinians to be purposeless, is revealed to be awash with purpose. There is a surprising amount of experimental support for the idea that cells (as well as genes) control inheritance, and this contradicts neo-Darwinism because the extra-nuclear cell contents are passed on unchanged during reproduction. It also provides the foundation for a creationist theory of baramin stasis. The concept of baramin stasis does not exist in secular biology, so creationists need to develop it.

In Part I of this article, I outlined the poverty of the Shannon theory of information as used in biology by evolutionists, and illustrated the 5-dimensional Gitt theory of information in biological terms. In Part II, I used the Gitt theory to redefine the ‘information challenge’ (where does the information come from in ‘goo to you’ evolution?) in creationist terms, showing that there is a vast gap in our knowledge of information storage, use and transfer in biology. Here, in Part III, I review experimental evidence on control of information during inheritance, and endeavour to develop a new perspective within a biblical framework.

How does biological information change?

As Darwinists struggle to find answers to the ‘information challenge’ using only the one-dimensional statistical view of information, creationists now have a powerful 5-dimensional argument to bring to bear on the problem. Here are two examples.

Antibodies and new enzymes

The human immune system can conjure up new antibodies (which are protein complexes) to deal in a very specific way with just about any foreign organic material that enters the body.1 Moreover, microbes can produce new enzymes (by changing existing enzymes) to metabolize synthetic organic molecules that did not exist prior to their manufacture by humans.2 Evolutionists have used both these lines of evidence to answer the ‘information challenge’ and argue that new information can arise by random mutations in existing biochemical pathways….

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